Caries draws its moves by bacteria that live in the mouth and living with the individual. This is normal commensals bacteria of the oral cavity that can not be eliminated as a precursor to many positive effects, just think even the simple competitiveness with other pathogenic species (for example, is so difficult that the diphtheria bacteria flourishes in the mouth because his penetration is prevented by the resident flora).
Our organization has evolved with these species of bacterial balance so purposes under normal conditions, their presence does not mean any harm as in other parts of the body (eg, for example, skin or intestine). But there are processes or conditions that may alter these purposes slatentizzare balances and the potential pathogen bacteria diners and the same occurs in the oral cavity with the onset of caries. The therefore infectious disease is a type of opportunist: in it for the changes of conditions, normally harmless bacteria causing the problem damage. The etiological caries therefore, as opportunistic infectious disease, because it complicates, rather than having to perhaps more than identify the type or types involved in appearance disease requires the specification of conditions in which bacteria become harmful.
The compromise pulp (Pulp) due process carioso is related to the destruction dentinal and therefore pulpari responses are mediated by the amount of residual dentine apparently healthy. For the therapy is important to diagnose the reversibility of ol irreversibility pathology. The pulp damage is limited and the flesh is capable of healing with appropriate therapies. The pulp pain in these cases is never spontaneous and continuous but only caused. The proper treatment restores homeostasis pulp and pulp remains alive and vital. In relation to the remaining dentin thickness of the treatments of choice are the simple restoration, the direct and indirect hooded and pulpotomia.